CARDIAC STIMULATION DEVICE

Brand Owner (click to sort) Address Description
EBR SYSTEMS EBR Systems, Inc. 480 Oakmead Parkway Sunnyvale CA 94085 Cardiac stimulation device, namely, cardiac pacemakers; cardiac resynchronization therapy system comprising a pacing device, catheter, sheath, electrode, battery and transmitter; medical device, namely, a device for synchronizing the contraction of the right and left sides of the heart; medical devices and apparatuses for stimulating the heart that incorporate implantable acoustic controller-transmitters, electrical sensors, and implantable acoustics receivers and stimulators, namely, cardiac pacemakers;SYSTEMS;
WISE EBR Systems, Inc. 480 Oakmead Parkway Sunnyvale CA 94085 Cardiac stimulation device, namely, cardiac pacemakers; cardiac resynchronization therapy system comprising a pacing device, catheter, sheath, electrode, battery and transmitter; medical device, namely, a device for synchronizing the contraction of the right and left sides of the heart; medical devices and apparatuses for stimulating the heart that incorporate implantable acoustic controller-transmitters, electrical sensors, and implantable acoustics receivers and stimulators, namely, cardiac pacemakers;
 

Where the owner name is not linked, that owner no longer owns the brand

   
Technical Examples
  1. An implantable cardiac stimulation device comprises a physiologic sensor and one or more pulse generators. The physiologic sensor is capable of sensing a physiologic parameter. The pulse generators can generate cardiac pacing pulses with a timing based on the physiologic parameter. The timed cardiac pacing pulses can prevent a sleep apnea condition. In one example, a cardiac stimulation device has a physiologic sensor and can be configured to pace a patient's heart according to a rest mode of operation. The cardiac stimulation device uses measurements from the physiologic sensor to prevent and treat sleep apnea using a revised rest mode of operation. The revised rest mode operates under a presumption that sleep apnea is primary to a reduced heart rate, rather than secondary, so that pacing at a rate higher than the natural cardiac rate during sleep will prevent sleep apnea.